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. 2021 Jan 6;14:4. doi: 10.1186/s12920-020-00848-0

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 5 — Altered methylation level of the imprinted genes between thoracic vascular malformation and control groups. a Hypermethylation of GRB10 gene between thoracic vascular malformation and control groups. b–f Hypomethylation of PEG10, INPP5F, NESP, MEG3 and KCNQ1OT1 respectively between thoracic vascular malformation and control groups. Compared with the normal group, 5 of the 18 imprinted genes in thoracic vascular malformation showed decreasing methylation modification including PEG10, INPP5F, NESP, MEG3 and KCNQ1OT1.TVM, thoracic vascular malformation. *P < 0.05; **P < 0.01