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. 2021 Jan 4;12(1):29. doi: 10.1038/s41419-020-03303-1

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — A–L HUVEC were pretreated with 0.5 µM KPT-8602 for 1 h and then infected with KSHV in the presence of KPT-8602 for 4, 12, 24, 36, and 48 h. Cells were collected for RNA extraction and examined for the expression of KSHV transcripts by RT-qPCR. M HUVEC were pretreated with 0.5 µM KPT-8602 for 1 h and then infected with KSHV in the presence of KPT-8602. The cell lysate was collected at 4, 12, 24, and 48 hpi and analyzed for the expression of viral proteins by Western blotting. β-actin served as the loading control.