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. 2020 Jun 2;24(1):31–44. doi: 10.1007/s10120-020-01092-2

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 11 — a P-STAT3 expression was higher in HPMCs treated with IL-17A than in the control. There was no increase in the expression of p-Smad2/3 in either the IL-17A-treated group or the control group. b E-cadherin expression was higher in HPMCs treated with IL-17A and Sttatic than those treated with IL-17A only. P-STAT3, Vimentin, and α-SMA expression was attenuated in HPMCs treated with IL-17A and Sttatic. c, d Densitometry analyses of p-STAT3 and α-SMA were performed in three independent experiments; data are expressed as the mean ± SD