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. 2021 Jan 5;12(1):9. doi: 10.1038/s41419-020-03329-5

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© The Author(s) 2021, corrected publication 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — A Prediction of the miRNA-9-5p target and B construction of the Thbs-2 mutation site. C The dual luciferase reporter assay (*P < 0.05, **P < 0.01 versus the WT group). D The immunoblotting and E quantitative data of Thbs-2 in astrocytes. F The immunofluorescence (bar = 100 μm) and G quantitative data of Thbs-2 in astrocytes (n = 4/group) (*P < 0.05, **P < 0.01, ***P < 0.001 versus the control group). H The immunofluorescence (bar = 100 μm) and I quantitative data of Thbs-2 in the cortex. J The immunofluorescence (bar = 100 μm) and K quantitative data of Thbs-2 in the hippocampus. L The immunofluorescence (bar = 100 μm) and M quantitative data of Thbs-2 in the SVZ (n = 5/group) (*P < 0.05, **P < 0.01, ***P < 0.001 versus the CCI group) (WT: wild type, Mut: mutant type, CCI: control cortex impact, SVZ: subventricular zone). Error bars indicate mean ± SD.