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. 2021 Jan 4;12(1):33. doi: 10.1038/s41419-020-03306-y

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — A, B Relative expression of miR-153-5p in tumor-normal tissues (A) and paired tumor-normal tissues (B) of TCGA. Horizontal lines represent the mean value. C–G Correlation of miR-153-5p with clinicopathological characteristics, including metastasis (C), TNM stage (D), lymph node metastasis (E), T stage (F), and histologic grade (G). H The expression level of miR-153-5p in ccRCC cell lines and human renal tubular epithelial cells. Horizontal lines in F, G represent the mean value. Horizontal lines in the boxes in C–E indicated the median value. Boxes span the interquartile range. Whiskers extend from the minimum to the maximum. The values in H are presented as mean ± SEM. I Kaplan–Meier analysis of the correlation between miR-153-5p and overall survival in ccRCC. J, K Gene Ontology (GO) enrichment analysis (J) and Kyoto Encyclopedia of Genes Genomes (KEGG) pathways enrichment analysis (K) for the expression of miR-153-5p. All experiments were conducted at least three independent times. *P < 0.05.