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. 2021 Jan 7;18:5. doi: 10.1186/s12984-020-00799-8

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 10 — Schematic representation of proposed neuromodulation of a cathodal tDCS over PMAs and b anodal tDCS over M1 related to posture and movement planning and preparation in individuals with stroke. In a, we hypothesized that cathodal tDCS over PMAs would down-regulate the hyperexcitability in this region and consequently restore the excessive inhibitory input (red dashed arrow) from PMAs to the PMRF via cortico-reticulospinal tract. In b, based on our findings, it is possible that application of anodal tDCS over M1 facilitates the subcortical PMRF via the cortico-reticular drive (green arrow) or direct excitations caused by the applied current