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. 2021 Jan 9;22:5. doi: 10.1186/s12865-020-00389-2

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 5 — Sera samples from WNV seropositive subjects lead to effective WNV neutralization but enhancement of ZIKV infection. Serum samples from WNV seropositive donors (symptomatic and asymptomatic) were assayed for enhancement and neutralization of ZIKV, WNV and DENV RVPs. a Serum dilution that causes peak ADE for respective RVPs was determined. b Serum dilution that causes 50% neutralization of respective RVPs was determined. c ADE for ZIKV, WNV and DENV RVPs at a serum dilution of 1:10. d Neutralization of respective RVPs at a serum dilution of 1:10