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. 2021 Jan 9;10(1):6. doi: 10.1038/s41389-020-00301-y

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© The Author(s) 2021, corrected publication 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — a GSEA analysis of the relationship between FAM83B levels and Wnt pathway. b Luciferase reporter assay of the effect of FAM83B expression on Wnt pathway activity. c Western blot analysis of nuclear β-catenin, c-myc, cyclin D1, BCL2, and cleaved Caspase-3 expression in ovarian cancer cells with FAM83B overexpression or knockdown. d Q-PCR analysis of MYC, TCF4, LEF1, FGF2, and CCND1 expression in ovarian cancer cells with FAM83B overexpression or knockdown. *p < 0.05. Error bars in panels are defined as s.d.