Table 2.
Environmental factors | Health outcomes |
---|---|
Pollution from traffic and industry (PM10, PM2.5, NO, NO2) | |
During childhood | Higher asthma prevalence after the school age |
During adulthood | Possibly higher asthma prevalence |
Lifelong |
Poorer lung function Higher rate of asthma exacerbations Conflicting results on AR onset |
Livestock farming (organic dust, toxins form microorganisms, gases like ammonia and methane) | Decreased lung function |
Black carbon | Possibly epigenetic changes leading to increased type two inflammation in children |
Interaction between air pollutants (PM10, nitrogen oxides) and allergens (pollen, fungal spores) | |
Production of more pollen, more allergens per pollen grain, and more PALMs per pollen grain |
Potentially, facilitation of IgE sensitization against aeroallergens Higher rate of asthma-related hospitalizations |