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. 2021 Jan 11;23:21. doi: 10.1186/s13075-020-02411-9

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 6 — Expression of TGFBR2 and IGF1R were altered following overexpression and knockdown of miR-320a. Normal human pulmonary fibroblasts were transfected with miR-320a mimics, miR-320a inhibitors, and scramble as control. a–c mRNA levels of TGFBR1, TGFBR2, and IGF1R were determined by SYBR Green real-time polymerase chain reaction analysis. d Protein was analyzed by western blot. Experiments were repeated at least 3 times. Values are means ± SD. *p < 0.05 versus scramble controls. **p < 0.01 versus scramble controls. ***p < 0.001 versus scramble controls