AzaA promoted the expression of VEGF and inhibited the expression of
TLR4, NF-kB, IL-1β, IL-6, and TNF-α. The immunohistochemically derived,
flap expression levels of VEGF, TLR4, NF-kB, IL-1β, IL-6, and TNF-α as
revealed by the DP2-TWAIN system (Olympus, Tokyo, Japan) operating at
400× magnification (a). The integral absorbances of flap VEGF, TLR4,
NF-kB, IL-1β, IL-6, and TNF-α levels in the control and treatment groups
(b). **P < 0.01, low-dose and high-dose AzaA groups
vs. control group; n = 6 per group. Arrows indicate
positive reaction. (A color version of this figure is available in the
online journal.)