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. 2021 Jan 12;21:51. doi: 10.1186/s12935-020-01715-9

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 3 — Knockdown of SNHG16 suppresses the migration and invasion of PC cells. a The migration ability of PC cells transfected with sh-SNHG16 or sh-NC was assessed by a wound healing assay. b The migration and invasion abilities of PC cells transfected with sh-SNHG16 or sh-NC were evaluated by transwell assays. c Western blot analysis of ICAM-1, VCAM-1 and MMP-9. β-Actin was used as the internal control. *P < 0.05, **P < 0.01