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. 2021 Jan 12;21:51. doi: 10.1186/s12935-020-01715-9

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 6 — MiR-302b-3p targets SLC2A4 and inhibits its expression directly. a Identification of the potential targets of miR-302b-3p using online prediction databases and qRT-PCR analysis. b The predicted binding site of miR-302b-3p within the SLC2A4 3’UTR. c The binding interaction between SNHG16 and miR-302b-3p was confirmed by luciferase reporter assay. d, e qRT-PCR and western blot analyses of the effect of miR-302b-3p on SLC2A4 expression. f, g Relative expression levels of SLC2A4 in PC tissues and cell lines. *P < 0.05, **P < 0.01, ***P < 0.001