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. 2021 Jan 7;40:101859. doi: 10.1016/j.redox.2021.101859

Fig. 1.

Fig. 1

FGF10 is significantly upregulated during hepatic IRI. (A) Protein expression levels of FGF10 in the livers of mice subjected to sham treatment or ischemia for 90 min followed by indicated periods of reperfusion (n = 4–6 mice/group). (B) Representative IHC staining of FGF10 in the livers of mice after hepatic ischemia for 90 min and reperfusion for 3, 6, and 24 h (n = 4–6 mice/group, magnification × 100). (C) Protein expression levels of FGF10 in major organ samples of mice subjected to sham treatment or 90 min of partial liver warm ischemia followed by 6 h of reperfusion (n = 4–6 mice/group). (D) Protein expression levels of FGF10 in primary hepatocytes and primary HSCs subjected to control or H/R stimulation. (E) The mRNA levels of Fgfr2b in primary hepatocytes and primary HSCs subjected to control or H/R stimulation. (F) Protein expression levels of FGF10 in L-02 and LX-2 cells subjected to control or H/R stimulation. (G) The mRNA levels of Fgfr2b in L-02 and LX-2 cells subjected to control or H/R stimulation. For (A), (C), (D), and (F), GAPDH was served as the loading control. All data are presented as mean ± SD, #P < 0.05. N.S.: non-significant; Con: control.