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. 2021 Jan 7;40:101859. doi: 10.1016/j.redox.2021.101859

Fig. 3.

Fig. 3

FGF10 overexpression alleviates liver damage and promotes AKT activation during hepatic IRI. Mice were subjected to 90 min of partial liver warm ischemia, followed by 6 h of reperfusion. (A) Representative H&E staining of liver sections from mice treated with AAV-GFP/AAV-Fgf10 after sham/IRI (magnification × 100). (B) Protein expression levels of AKT signaling in the livers of mice treated with AAV-GFP/AAV-Fgf10 after sham/IRI. (C) Protein expression levels of MEK/ERK signaling in the livers of mice treated with AAV-GFP/AAV-Fgf10 after sham/IRI. (D) Protein expression levels of JNK and p38 signaling in the livers of mice treated with AAV-GFP/AAV-Fgf10 after sham/IRI. For (B–D), GAPDH was served as the loading control. All data are presented as mean ± SD, n = 4–6 mice/group, #P < 0.05. N.S.: non-significant.