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. 2021 Jan 14;21:33. doi: 10.1186/s12906-020-03196-9

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 3 — The effect of luteolin on LPS-induced hydrogen peroxide production, mast cell degranulation and intestinal microcirculation blood flow. a The effect of luteolin on LPS-induced hydrogen peroxide production. Pictures were captured at 400x magnification. Arrows: DHR fluorescence on the venular walls. b The effect of luteolin on LPS-induced mast cell degranulation. Pictures were captured at 400x magnification. Arrows: degranulated mast cell. c The effect of luteolin on LPS-stimulated intestinal microcirculation blood flow. All of the representative images of each group were presented with the respective quantification showing right correspondingly. All of the data were expressed as means ± SEM (n = 6). Significant differences were indicated by ^##P< 0.01 as compared with control group, ^*P< 0.05, ^**P< 0.01 as compared with LPS group