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. 2020 Oct 29;40(2):277–291. doi: 10.1038/s41388-020-01525-3

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — A Overexpression efficiency of LINC00261 in the pancreatic cancer cell lines Panc1 and Mia-PaCa2. Cell proliferation capacity of pancreatic cancer was measured by B CCK8 assay, C colony formation assay, and D EdU assay using Panc1 and Mia-PaCa2 cells. The glycolysis levels of Panc1 and Mia-PaCa2 cells were evaluated by E ECAR and F OCR, which were detected using a seahorse analyzer, and by G glucose uptake and H lactate production. The data are presented as the mean ± SD of three independent experiments. *P < 0.05; **P < 0.01; ***P < 0.001.