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. 2020 Dec;8(24):1674. doi: 10.21037/atm-20-6989

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2020 Annals of Translational Medicine. All rights reserved.

Open Access Statement: This is an Open Access article distributed in accordance with the Creative Commons Attribution-NonCommercial-NoDerivs 4.0 International License (CC BY-NC-ND 4.0), which permits the non-commercial replication and distribution of the article with the strict proviso that no changes or edits are made and the original work is properly cited (including links to both the formal publication through the relevant DOI and the license). See: https://creativecommons.org/licenses/by-nc-nd/4.0.

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MicroRNA-30s modulate Cx43 expression in STZ-induced DN rats. Rats were divided into four groups: control, STZ, STZ + microRNA-30 family mimics, and STZ + microRNA-30 family inhibitors. (A) RT-qPCR analyses of microRNA-30s among the four groups. (B) IHC analyses of Cx43 protein expression among the four groups (×40). Representative images from 4 mice in each group are shown. (C) Quantitative analyses of the IOD of Cx43 are shown in the bar graph, and data are expressed as the mean ± SEM of three experiments. ^#, P<0.05 vs. the control group. *, P<0.05 vs. the STZ group. STZ, streptozotocin; DN, diabetic nephropathy; IHC, immunohistochemistry.