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. 2020 Dec;8(24):1674. doi: 10.21037/atm-20-6989

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2020 Annals of Translational Medicine. All rights reserved.

Open Access Statement: This is an Open Access article distributed in accordance with the Creative Commons Attribution-NonCommercial-NoDerivs 4.0 International License (CC BY-NC-ND 4.0), which permits the non-commercial replication and distribution of the article with the strict proviso that no changes or edits are made and the original work is properly cited (including links to both the formal publication through the relevant DOI and the license). See: https://creativecommons.org/licenses/by-nc-nd/4.0.

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MicroRNA-30s modulate podocyte injury in vivo. Rats were divided into four groups: control, STZ, STZ + microRNA-30 family mimics, and STZ + microRNA-30 family inhibitors. (A) Representative photomicrographs (original magnification, ×400) prepared from PAS-stained kidney sections. (B) The glomerular sclerosis scores of renal tissues among the four groups. (C) Quantitative analyses of the IOD of podocin are shown in the bar graph, and the data are expressed as the mean ± SEM of three experiments. (D) IHC analyses of podocin protein expression among the four groups (×40). Representative images from 4 mice in each group are shown. ^#, P<0.05 vs. the control group. *, P<0.05 vs. the STZ group. STZ, streptozotocin; PAS, Periodic Acid-Schiff stain; IHC, immunohistochemistry.