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. 2021 Jan 18;18(3):660–674. doi: 10.1038/s41423-020-00627-y

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — Inhibitory activity of GPI-anchored antibodies in transduced human CD4+ T cells during HIV-1 infection. CEMss-CCR5 cells transduced with GPI-m36.4/GFP or GPI-FluIgG03/GFP were infected with 1,000 TCID₅₀ of NL4-3 (A), SG3.1 (B), MJ4 (C), or RHPA.c/2635 (D), and intracellular HIV-1 P24 Gag and GFP expression was monitored over time by flow cytometry. Data from an experiment representative of three independent experiments are shown