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. 2021 Jan 18;12(1):97. doi: 10.1038/s41419-020-03340-w

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — A, B Knockdown efficiency of MEDAG in MCF-7 and MDA-MB-467 cell lines. C MEDAG knockdown inhibited breast cancer cell proliferation in MCF-7 (up) and MDA-MB-468 (down) cells. Cell proliferation was measured by a CCK-8 assay. D Transwell assays showing that MEDAG silencing decreased cell invasion in MCF-7 (up) and MDA-MB-468 (down) cells. Quantitative analysis of the invasion ratio is shown. E Wound-healing assay showing that MEDAG knockdown reduced cell migration. Representative images are shown at 0 and 48 h. The values represent mean ± SD of three independent experiments. **P < 0.01, ***P < 0.001, ****P < 0.0001 vs the control group.