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. 2021 Jan 18;11:1688. doi: 10.1038/s41598-021-81179-7

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Reduction in myofibroblasts, vascularity, and dermal cellularity in MSTC-treated wounds. Representative images of immunohistochemical staining for α-smooth muscle actin (a, myofibroblasts highlighted by arrows, scale bar = 25 μm) and von Willebrand factor (b, vascular structures highlighted by arrows, scale bar = 50 μm). MSTC-treated wounds had reduced densities of α-SMA + myofibroblasts after 2 weeks (c), as well as vWF + blood vessels at both time points (d). Overall cellularity in the dermis was significantly elevated in control wounds at 2 weeks, compared to both MSTC-treated wounds and unwounded normal skin (e). *p $\leq$ 0.05, **p $\leq$ 0.005, ***p $\leq$ 0.0005, ****p $\leq$ 0.0001.