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. 2021 Jan 19;13:7. doi: 10.1186/s13098-021-00625-8

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 6 — Overexpression of miR-93-5p alleviated LPS-induced cell injury in HK2 cells by targeting OXSR1. a The protein level of OXSR1 in HK2 cells transfected with pcDNA or OXSR1 was measured by western blot. b–h LPS-treated HK2 cells were transfected with miR-NC, miR-93-5p, miR-93-5p+pcDNA or miR-93-5p+OXSR1. b The protein level of OXSR1 in LPS-induced HK2 cells after transfection was tested by western blot. c Cell viability in these cells was assessed by CCK-8 assay. d, e Cell apoptosis and percentage of cells at different stages were monitored by flow cytometry assay. f The concentration of IL-1β and TNF-α was examined by ELISA using corresponding kits. g, h The levels of SOD and MDA in these cells were examined by corresponding specific kits. *P < 0.05, **P < 0.01, ***P < 0.001, ****P < 0.0001