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. 2021 Jan 12;14(1):e007022. doi: 10.1161/CIRCHEARTFAILURE.120.007022

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© 2020 The Authors.

Circulation: Heart Failure is published on behalf of the American Heart Association, Inc., by Wolters Kluwer Health, Inc. This is an open access article under the terms of the Creative Commons Attribution Non-Commercial-NoDerivs License, which permits use, distribution, and reproduction in any medium, provided that the original work is properly cited, the use is noncommercial, and no modifications or adaptations are made.

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Figure 3. — Differences in upregulated proteins between patients with hypertrophic cardiomyopathy sarcomere mutation-positive (HCM_SMP) and sarcomere mutation-negative (HCM_SMN). Protein interaction cluster of proteins that are only significantly upregulated for the HCM_SMP vs nonfailing interventricular septum (NF_IVS) or the HCM_SMN vs NF_IVS comparison were identified and are displayed with the most significant corresponding gene ontology (GO) term. The top 5 protein interaction clusters of upregulated proteins are displayed The color gradient from light to dark indicates an increase in fold change.