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. 2021 Jan 21;12(1):109. doi: 10.1038/s41419-020-03385-x

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 7 — A Infiltrated macrophages (F4/80) in UUO and ADR mice exhibited different autophagy levels (LC3) (400×). B Primary BMDMs (M0, M1, and M2) were treated with or without VEGF-C, and the protein expression of autophagy-related markers (LC3 and p62) was measured by western blotting. C Immunofluorescence was used to show the effect of VEGF-C on autophagy in BMDM subtypes (400×). D, E M1 cells were treated with VEGF-C, siRNA targeting VEGF-C (Si-VEGF-C) and/or the VEGFR3 inhibitor SAR131675 (SAR-23/46 nM), and the protein expression of autophagy markers (LC3 and p62) was measured by western blotting. N = 6/group.