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. 2021 Jan 21;12(1):106. doi: 10.1038/s41419-021-03410-7

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — A, B and C show the result of survival analysis with all patients, patients receiving EGFR targeted therapy (TKI group), and no EGFR targeted therapy (non-TKI group). All molecular features fall into two categories, dichotomous and continuous: gender(male/female), pathology (LUAD/other), metastatic type (synchronous/metachronous), EGFR TKI therapy (with TKI/non-TKI), EGFR (positive/negative), TP53 (positive/negative), ARID1B (positive/negative), ZFHX3 (positive/negative), MLL3 (positive/negative) and PD-L1 (positive/negative) were performed Kaplan-Meier survival analysis. For continuous factors, maximally selected rank statistics was performed to calculate optimal cutoffs. Those factors were divided into higher and lower groups according to cutoffs. Hazard ratio (HR) was calculated by high group compare to low group.