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. 2021 Jan 22;12:77. doi: 10.1186/s13287-021-02156-5

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 7 — The low expression of hsa-miR-11401 inhibited its protective effects. a The mode pattern that collecting extracellular vesicles from inhibitor-pretreated hP-MSCs and the flow that EV^inhibitor repair the injured hUC-MSCs. b The expression of hsa-miR-11401 in EVs and inhibitor-contained EVs, ^*P < 0.05 versus EV group. c The expression of SCOTIN in Blank, EV, and EV^inhibitor groups, ^*P < 0.05 versus EV groups. d The morphology of hUC-MSCs in Blank, EV, and EV^inhibitor groups. Scale bars, 100 μm. e The expression of cleaved-caspase 3 in Blank, EV, and EV^inhibitor groups. Scale bars, 20 μm