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. 2021 Jan 20;14:265–277. doi: 10.2147/DMSO.S280765

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© 2021 Wang et al.

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EPB41L4A-AS1 knockdown increased glycolysis and mitochondrial stress. (A) ECAR in shnc and shEPB41L4A-AS1 cells (n=3). (B) OCR in shnc and shEPB41L4A-AS1 cells (n=3). (C) Schematic model of MYD88-mediated activation of the NF-κB pathway by EPB41L4A-AS1 in T2DM-related inflammation. Data are shown as mean ± SD. *P<0.05, **P<0.01; Student’s t-test.