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. 2021 Jan 8;11:587140. doi: 10.3389/fphar.2020.587140

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Copyright © 2021 Murru, Carta, Manca, Sogos, Pistis, Melis and Banni

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Concentrations of palmitoylethanolamide (PEA) and oleoylethanolamide (OEA) analyzed by LC-MS as described in (Piras et al., 2015), in rat horizontal slices containing the midbrain incubated for 1 h with 100 µM of synthetic and endogenous ligands of PPARα or their vehicle: agonist WY14643 (WY), oleic acid (18:1), palmitic acid (16:0), linoleic acid (18:2), c9-t11, t10-c12, and a mixture of both CLA isomers (CLA), or PPARα antagonist (MK886). Error bars represent SD; n = 6. * denote significant differences (p < 0.05), vs. control (one-way ANOVA).