Figure 2.

Venous congestion and glomerular haemodynamics. Renal venous congestion caused by intravascular congestion or an increase in intra‐abdominal pressure can lead to a build‐up of renal interstitial hydrostatic pressure through peritubular capillary congestion and development of interstitial oedema. This presumably leads to increased efferent arteriolar pressure, which may initially result in glomerular hyperfiltration; however, further increases in renal venous pressure with decreases in renal perfusion pressure ultimately reduce glomerular filtration rate.