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. 2021 Jan 26;22:48. doi: 10.1186/s13059-021-02259-5

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 3 — a The distribution of AS event type in tumor-specific AS events, comparing primary and recurrent GBMs. b The fractions of tumor-specific AS events in cell-surface proteins, compared between primary and recurrent GBM. c The percentages of tumor-specific neojunctions found in different protein domains. d The frequencies of tumor-specific neojunctions in GBM neoplastic cells from Smart-seq2 scRNA-seq. e The fractions of cases with at least one CPTAC-confirmed neojunction. f The average number of CPTAC-confirmed neojunctions per case