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. 2021 Jan 25;12:85. doi: 10.1186/s13287-021-02154-7

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 4 — MiR-672 regulates vascularization in vivo. a Micro-CT scanning of newly regenerated blood vessels in the defect regions repaired by CPC scaffold seeded with the ADSCs/Lenti-miR-672, ADSCs/Lenti-as-miR-672, and ADSCs/Lenti-miR-NC groups (within the white circle). b Histological observation of newly formed blood vessels in the regenerated bone tissue indicated by the perfused Microfil (black arrow). c Analyses of the blood vessel volume, percentage of blood vessel volume/total volume, and Microfil-labeled blood vessel number in the respective groups. Statistical significances were determined by unpaired Student’s t test using GraphPad Prism Version 8. *P < 0.05, **P < 0.01, ***P < 0.001