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. 2020 Dec 17;102(1):22–31. doi: 10.1111/iep.12367

Figure 3.

Figure 3

MiR‐17‐5p was the target of circ‐ITCH and promoted apoptosis of H2O2‐induced myocardial cells. A and B, Binding information was acquired from bioinformatics tools (TargetScan) and proved through luciferase report assay, P < .05. C, Expressions of miR‐17‐5p in H9c2 cells treated by H2O2 were measured with RT‐qPCR, P < .05. D, Suppressed miR‐17‐5p expression was checked through RT‐qPCR, P < .05. E, Cell viabilities were analysed by CCK‐8, P < .05. F, ATP concentrations were detected, P < .05. G, Apoptosis rate with suppressed miR‐17‐5p was measured with flow cytometry, P < .05. H, Caspase 3, p53 and PARP levels were detected by Western blot, P < .05. Student's t test and one‐way ANOVA were used