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. 2021 Jan 28;13:5. doi: 10.1186/s13099-021-00401-z

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 2 — Chronic AIEC LF82 and C. albicans administration induced intestinal inflammation in DSS-induced chronic colitis in mice. Colonic gene expression of: a proinflammatory cytokines Il1b, Il6, Il12b, Il17, Ifng and chemokine Ccl4; b immune cell recruitment markers Adgre1, Ly6g and Cd3e, and T-cell transcription factors Tbx21, Gata3, Rorc and Foxp3. Results correspond to fold-increase when compared to mice receiving drinking water and are expressed as means ± SEM; *P < 0.05; **P < 0.01; ***P < 0.001 for colonized vs. non-colonized mice receiving DSS only (Mann–Whitney U test)