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Proposed model for baicalein as an inhibitor to suppress the cell proliferations in SiHa and HeLa cells. Baicalein decreased the expression of p-GSK3β via the downregulation of p-AKT, thereby enhancing the activity of GSK3β. GSK3β phosphorylated cyclin D1, promoting the degradation of cyclin D1, which further arrested cells at the G0/G1 phase, inhibiting the proliferation of SiHa and HeLa cervical cancer cells.
