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. 2021 Jan 13;6(1):e01156-20. doi: 10.1128/mSphere.01156-20

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Copyright © 2021 Naha et al.

This is an open-access article distributed under the terms of the Creative Commons Attribution 4.0 International license.

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FIG 2 — Schematic presentation of MGEs associated with bla_OXA-181/232 and bla_NDM-5 in the K. pneumoniae strains isolated from neonates. Heterogeneity of the genetic environment found in the studied carbapenemases: (a) bla_OXA-181/232 (Kp2-Kp4, Kp6-Kp11), (b) bla_OXA-181 in Kp1, and (c) genetic environment of transposon 125 (Tn125) harboring bla_NDM-5 (Kp3, Kp9-Kp11). Genes and their corresponding transcription orientations are indicated by horizontal arrows. Target site duplications (ATATA) generated by the insertion of Tn2013 are indicated by white triangles. mobA, mobB, mobC, and mobD, mobilization relaxosome proteins; ΔlysR, truncated LysR-type transcriptional regulator; ΔereA, truncated erythromycin esterase; repA, replicase; tnpA, transposase; IS, insertion sequence; ble_MBL; bleomycin resistance gene; trpF, N-(5′-phosphoribosyl) anthranilate isomerase; tat, twin-arginine translocation pathway signal sequence protein; Hypo. protein, hypothetical protein; Δ, denotes deletion or truncation.