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. 2020 Dec 8;11:607605. doi: 10.3389/fphar.2020.607605

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Copyright © 2020 Li, Yang, Chen, Guo, Gao, Wang, Duan and Li.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Knockdown of MOF promoted the apoptosis of thyroid cancer cells. (A,B) In BHP-10-3 and TT2609 cell lines, the effect of knockdown MOF were measured using PI/FITC staining assay. (C,D) Western blot assay was performed to detect the protein expression of apoptosis marks including caspase 3, cleaved-caspase 3, BAX, BCL2 in stable MOF-knockdown and control BHP-10-3 and TT2609 cells. MOF regulated TNK2 transcriptional expression in thyroid cancer cells.