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. 2021 Jan 19;17(1):e1009287. doi: 10.1371/journal.pgen.1009287

Table 1. Abl mutant MARCM clones.

(A) Ablmut/ Ablmut MARCM clones.
Neuroblast clones
Genotype WT Absence of α lobe Absence of β lobe Absence of α/β lobes n
Control 22
100%
-- -- -- 22
Abl2 / Abl2 43
90%
5
10%
-- -- 48
Abl4 / Abl4 37
82%
5
11%
3
7%
-- 45
Abl1 / Abl1 71
92%
3
4%
3
4%
-- 77
(B) Visualization Abl2/Abl4 MARCM clones.
Two-cells / Single-cell clones
Genotype WT α branch growth defect β branch growth defect α/β branch growth defects n
Control 30
100%
-- -- -- 30
Abl2 / Abl4 2
13%
10
67%
-- 3
20%
15
>Two-cells clones
Genotype WT Absence of α lobe Absence of β lobe Absence of α/β lobes n
Control 9
100%
-- -- -- 9
Abl2 / Abl4 6
23%
14
54%
2
8%
4
15%
26
Neuroblast clones
Genotype WT Absence ofα lobe Absence of β lobe Absence of α/β lobes n
Control 9
100%
-- -- -- 9
Abl2 / Abl4 2
17%
7
58%
-- 3
25%
12

WT: wild-type clones. n: number of clones analyzed.

Full genotypes: A) Control: y w67c23 hs-FLP122 / +; c739-GAL4 UAS-mCD8-GFP / +; tubP-GAL80, FRT2A / FRT2A. Mutant:. y w67c23 hs-FLP122 / +; c739-GAL4 UAS-mCD8-GFP / +; tubP-GAL80, FRT2A / Abl2 FRT2A. y w67c23 hs-FLP122 / +; c739-GAL4 UAS-mCD8-GFP / +; tubP-GAL80, FRT2A / Abl4 FRT2A. y w67c23 hs-FLP122 / +; c739-GAL4 UAS-mCD8-GFP / +; tubP-GAL80, FRT2A / Abl1 FRT2A. B) Control: w* tubP-GAL80 hs-FLP122 FRT19A / w* sn FRT19A; c739-GAL4 UAS-mCD8-GFP / UAS-mCD8-GFP. Abl2/Abl4: w* tubP-GAL80 hs-FLP122 FRT19A / w* sn FRT19A; c739-GAL4 UAS-mCD8-GFP / UAS-mCD8-GFP; Abl2 / Abl4.