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. 2021 Jan 29;5:1. doi: 10.1038/s41538-021-00084-5

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 7 — a–e Expression levels of inflammatory mediators in hippocampus (hpc), cerebral cortex (ctx), spleen, and small intestine determined by qRT-PCR. Il-1b (a), Tnf (b), Ccl5 (c), Cxcl13 (d), and Hmgb1 (e). f–g Expression levels of Tlr2/4 in hpc, ctx, and hypothalamus (hypo) determined using qRT-PCR. All mRNA species were quantified relative to the expression of Gapdh and presented as fold changes compared to control (Cont). Data are shown as means ± standard error. Cont, n = 8; RA, n = 9, *p < 0.05, ^#p < 0.10.