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Wnt/β-catenin pathway was the mechanism by which TMEM168 exerted its oncogenic activities in human GBM cells. siTMEM168 induced changes in (A) protein levels of β-catenin, C-myc, cyclin D1, and survivin, and (B) proliferative changes were significantly reversed by a Wnt/β-catenin activator LiCl in U87 cells. **p < 0.01 versus siNC; ##p < 0.01 versus siTMEM168.
