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. 2020 May 12;37(1):35–49. doi: 10.1007/s10565-020-09525-5

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 6 — The associations among the expression of sf-RON, RON, β-catenin and SIX1, and survival of gastric cancer patients. a Representative images of biopsies containing the expression of β-catenin and SIX1 in gastric cancer patient tissues with low and high expression of sf-RON and RON (× 400). b, c Correlation of sf-RON and RON expression with β-catenin and SIX1. d–g Kaplan-Meier OS survival curves (log-rank tests) and PFS survival curves (log-rank tests) of patients with varying sf-RON, RON, β-catenin, and SIX1 expression. h Schematic model showing the role of sf-RON/β-catenin/SIX1 signaling axis in the regulation of cell proliferation and glucose metabolism in the GC cells