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. 2021 Feb 1;9:19. doi: 10.1186/s40478-021-01119-4

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 4 — Nlgn1 reduction in frontal grey matter of tauopathies. Nlgn1 levels analysed in a tauopathies cohort including AD (n = 10), PiD (n = 9), CBD (n = 10), PSP (n = 11) and Ctrl (n = 10) from frontal grey matter. a representative Western blot and relative GAPDH signal. Samples were run as singlicates. A calibration curve of recombinant Nlgn1 fusion protein was used to estimate the amount of Nlgn1 present in the samples (as above). b The bars presented in the scatterplots show the median and the interquartile range. Significance: p ≤ 0.05