Fig. 3. Stroop performance and relationships with [11C]-(+)-PHNO BPND and DMN engagement.
a Incongruent errors and interference delays (incongruent minus congruent reaction times) did not differ between cocaine-use disorder (CUD) and healthy comparison (HC) participants. Error bars indicate standard deviation. b In HC participants, higher D2R-related binding in the dorsal putamen (DPU) was associated with shorter interference delays (DPU and delay residuals from regressions with BPND in the substantia nigra (SN) to reflect results of mixed model analyses). c In CUD, higher D3R-related binding in the SN was associated with shorter interference delays (SN and delay residuals from regressions with BPND in the DPU to reflect results of mixed model analyses). d In HC participants, greater average DMN engagement (i.e., less DMN suppression) was associated with reduced interference delays. *P < 0.05; **P < 0.01.