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. 2021 Jan 20;7:610561. doi: 10.3389/fcvm.2020.610561

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Copyright © 2021 Zhang, Shen, Shi and Yu.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Regulatory mechanisms of miR-223-3p in myocardial infarction. MiR-223-3p participates in different pathophysiological activities affecting myocardial infarction through regulation on multiple genes and diverse signaling pathways. MiR-223-3p promotes hypoxia-induced cardiomyocyte injury and inhibits the recovery of myocardial ischemia with its anti-angiogenesis effects during myocardial infarction. Myocardial fibrosis and arrhythmia can be induced by miR-223-3p to impact the prognosis of myocardial infarction. Besides, miR-223-3p is helpful for alleviation of I/R injury which often occurs after myocardial infarction.