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. Author manuscript; available in PMC: 2022 Jan 1.
Published in final edited form as: Behav Genet. 2020 Sep 21;51(1):30–44. doi: 10.1007/s10519-020-10017-9

Table II.

Genetic and environmental stability of stress variables across waves

Bivariate Decomposition rA rE Biv A Biv E Pheno r
Age 23 Dependent-Age 29 Dependent .94* [.15] .07 [.09] .28* [.06] .05 [.06] .32* [.05]
Age 23 Dependent LT-Age 29 Dependent LT .78* [.22] −.03 [.17] .29* [.09] −.02 [.10] .28* [.08]
Age 23 Independent-Age 29 Independent 1.0 [.87] .20 [.13] .05 [.07] .17 [.11] .22* [.08]
Age 23 Independent LT-Age 29 Independent LT .55* [.17] .01 [.18] .29* [.09] .00 [.08] .29* [.06]

Shared environment (C) correlations were not estimated in decompositions but these same decompositions without dropping all other C parameters are reproduced in Supplemental Table SVI. rA=genetic correlation; rE=nonshared environmental correlation; BivA= bivariate heritability—the phenotypic correlation explained by the genetic correlation; Biv E= bivariate nonshared environmentality—the phenotypic correlation explained by the nonshared environmental correlation; 23 and 29 indicate mean ages of assessment; LT=lifetime stress variable. For each pair of variables, Biv A + Biv E equals the model phenotypic correlations, which are very close to those in Supplemental Table SIII (significance of Pheno r determined by Table III for consistency).

*

p< 0083, boldface p< .05; determined with chi-square difference tests dropping the relevant Cholesky cross-paths, but bootstrapped standard errors shown in brackets for reference. When standard errors disagree with chi-square difference tests, we deemed significance from chi-square difference tests.