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. 2021 Jan 25;2021:8863789. doi: 10.1155/2021/8863789

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Copyright © 2021 Rodrigo Carrasco et al.

This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Potential cardioprotective therapies for anthracycline-induced cardiotoxicity. ACE: angiotensin-converting enzyme; CBR3: carbonyl reductase 3; DHA: docosahexaenoic acid; DNA DSBs: indicates deoxyribonucleic acid double-stranded breaks; EPA: eicosapentaenoic acid; GPx: glutathione peroxidase; MnSOD: Mn-superoxide dismutase; MPO: myeloperoxidase; n-3 PUFAs: n-3 polyunsaturated fatty acids; Nrf2: nuclear factor erythroid 2-related factor 2; PGC1 α/β: peroxisome proliferator-activated receptor-γ coactivator 1-α, and 1-β; PTP: permeability transition pores; Rac1: a subunit of NADPH oxidase; ROS: reactive oxygen species; Top2β: topoisomerase 2β; UCP2: mitochondrial uncoupling protein 2.