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The mechanism by which neutrophils exacerbate acute pancreatitis (AP). The release of CXCL2 in early AP tissues induces the recruitment of neutrophils. Activated neutrophils induce the release of granulocytes, NET formation, and macrophage recruitment by releasing inflammatory chemokines, leading to increased pancreatic injury. Some of the activated neutrophils reverse migrate to the circulatory system, causing acute lung injury.
