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. 2021 Feb 3;12:757. doi: 10.1038/s41467-021-21026-5

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — a The number of nonsynonymous (NS) mutations per tumor. b The potential chordoma driver genes. c The middle gene panel reports NS mutations in known cancer driver genes that were detected in this study. SNV single nucleotide variant, SV structural variant, HOMO DEL homozygous deletion, AMP amplification, Germline DUP germline duplication. d The recurrence status (no: gray; yes: red), age (from younger to older: blue to yellow), sex (female: red; male: blue), and SCNA group (see Fig. 3). See also Supplementary Fig. 2.