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. 2021 Jan 4;45(3):921–932. doi: 10.3892/or.2021.7920

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Copyright: © Ji et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 6. — CDCA5 knockdown inhibits prostate tumour growth in vivo. (A and B) Volumes of PC-3 subcutaneous xenograft tumours were compared between the shCDCA5 and shCON groups (n=6, *P<0.05). (C) Tumour weights were compared between the shCDCA5 and shCON groups (n=6, *P<0.05). (D) Representative images of the protein expression levels of CDCA5, p-ERK, Ki67 and cleaved caspase-3 in xenograft tumours (n=6). (E) Western blot analysis of CDCA5, ERK, p-ERK, Ki67 and cleaved/pro caspase-3 expression levels in the tumours from the shCDCA5 and shCON groups. GAPDH was used as an internal control. CDCA5, cell division cycle-associated 5; sh, short hairpin; CON, control; p-, phosphorylated; ERK, extracellular signal-regulated kinase.