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. 2021 Feb 3;21:87. doi: 10.1186/s12935-020-01706-w

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 7 — HMGA2 was high-expressed in colon cancer, and was negatively correlated with miR-532-3p and positively correlated with LINC00963. a TargetScan7.2 and (B-C) dual-luciferase reporter gene assay predicted and verified that HMGA2 binds to miR-532-3p. d HMGA2 was high-expressed in CRC. e–h The correlation between the expression of LINC00963, miR-532-3p and HMGA2 was analyzed by Pearson. ^△△△P < 0.001 vs Blank; ^&&&P < 0.001 vs Normal